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dc.contributor.authorWere, T.
dc.contributor.authorMaina, Naomi
dc.contributor.authorNgotho, Joseph Maina
dc.contributor.authorThuita, John Kibuthu
dc.contributor.authorMwangangi, David Mumo
dc.contributor.authorKagira, John Maina
dc.contributor.authorNdung'u, Joseph Mathu
dc.contributor.authorSternberg, Jeremy
dc.date.accessioned2013-11-13T08:17:03Z
dc.date.available2013-11-13T08:17:03Z
dc.date.issued200-05
dc.identifier.citationInfect Immun. 2004 May; 72(5): 3063–3065.en_US
dc.identifier.urihttp://ir-library.ku.ac.ke/handle/123456789/7614
dc.descriptiondoi: 10.1128/IAI.72.5.3063-3065.2004en_US
dc.description.abstractA vervet monkey model of trypanosomiasis was used to study inflammatory cytokine responses in serum and cerebrospinal fuid (CSF). Gamma interferon levels were transiently up-regulated in serum between days 6 and 8 of infection, followed by a sustained up-regulation of tumor necrosis factor alpha (TNF-α) and soluble TNF receptor 1. At no time were these cytokines detectable in the CSF.en_US
dc.language.isoenen_US
dc.publisherInfection and Immunityen_US
dc.titleProinflammatory Cytokine Expression in the Early Phase of Trypanosoma brucei rhodesiense Infection in Vervet Monkeys (Cercopithecus aethiops)en_US
dc.typeArticleen_US


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